Dr. Jennifer H. Shaw received her BS in Biological Sciences in 1995 from Florida State University in her hometown of Tallahassee. In 2000, she earned her PhD in Pharmacology from The University of Montana School of Pharmacy where she conducted her dissertation research on C. trachomatis antigen-pulsed dendritic cells as an NIH pre-doctoral fellow at the nearby Rocky Mountain Laboratory, a branch campus of the National Institute of Allergy and Infectious Disease. Dr. Shaw completed her postdoctoral research in vascular biology at Oklahoma State University in The College of Veterinary Health Sciences studying the role of hydrogen peroxide in the regulation of angiogenic factors. Since 2005, she has been teaching medical physiology and pharmacology to a wide spectrum of students including undergraduates, MS and PhD students, second-year veterinary students, and currently first- and second-year DO students.
Dr. Shaw's research background includes the use of: i) a murine model to study the pathogenesis of and immune response to C. trachomatis, ii) in vitro assays as well as an extremophile fish model to study the oxidative vascular mediators hydrogen peroxide and hydrogen sulfide, respectively. Dr. Shaw has mentored both PhD and MS students as well as several undergraduate research scholars. Her basic science research is currently funded by the National Institutes of Health and has been previously funded by the National Science Foundation. Dr. Shaw has published in journals such as Proceedings of the National Academy of Sciences of the United States of America, The American Journal of Physiology, Frontiers in Cellular & Infection Microbiology, Comparative Biochemistry & Physiology, Microcirculation, Microvascular Research, and Infection & Immunity.
Dr. Shaw's research background includes the use of: i) a murine model to study the pathogenesis of and immune response to Chlamydia trachomatis, ii) in vitro assays as well as an extremophile fish model to study the oxidative mediators hydrogen peroxide and hydrogen sulfide, which are both relevant in vascular biology. These research projects have been funded by the National Institutes of Health and National Science Foundation.
Lutter E and Shaw JH. The role of Chlamydia trachomatis Incs in modulating the immune response. NIH, R15 AREA, total $441,393 (Role: Co-PI, $67,110), Mar 2020 - Feb 2023.
Lutter E and Shaw JH. The role of CT228 in Chlamydia trachomatis pathogenesis. NIH, R15 AREA, total $426,916 (Role: Co-PI, $213,458), Jul 2015 - Jun 2019.
Tobler M, Shaw JH, Kelley J. Physiological Adaptation to Extreme Environments: Genes, Function, and Evolutionary Patterns. NSF-IOS Integrative Ecological Physiology, total $800,005 (Role: Co-PI, $150,765), Mar 2016 - Mar 2019.
Shaw JH. The role of angiogenic factors in the development of atherosclerosis during Chlamydia pneumoniae infection. Oklahoma Center for Respiratory & Infectious Diseases, NIH-CoBRE, total $50,000 (Role: PI, $50,000), Jul 2015 – Jun 2016.
Shaw, JH. Advancing the Physiology Curriculum with iWorx animal and human physiology lab teaching kits. Oklahoma State University Arts & Sciences Student Tech Fee Program (Role: PI, $39,440), 2013 – 2014.
Shaw, JH. Development of a Shared Tissue Culture Facility. Oklahoma State University VP for Research & Technology Transfer Funds (Role: PI, $59,749), 2012-2013.
Shaw, JH. The Effects of Shear Stress on Endogenous Production of Hydrogen Sulfide by Human Coronary Artery Smooth Muscle Cells, Oklahoma State University (Role: PI, $6,500), 2013.
Shaw, JH. Effect of hyperlipidemia on signaling pathways mediating exercise-induced vascular remodeling. Research Advisory Committee (RAC) Award, College of Veterinary Health Sciences, Oklahoma State University (Role: PI, $9,947), 2010-11.
© 2024 Philadelphia College of Osteopathic Medicine