Dr. Shaw's research background includes the use of: i) a murine model to study the
pathogenesis of and immune response to Chlamydia trachomatis, ii) in vitro assays
as well as an extremophile fish model to study the oxidative mediators hydrogen peroxide
and hydrogen sulfide, which are both relevant in vascular biology. These research
projects have been funded by the National Institutes of Health and National Science
Lutter E and Shaw JH. The role of Chlamydia trachomatis Incs in modulating the immune response. NIH, R15 AREA, total $441,393 (Role: Co-PI, $67,110), Mar 2020 - Feb 2023.
Lutter E and Shaw JH. The role of CT228 in Chlamydia trachomatis pathogenesis. NIH, R15 AREA, total $426,916 (Role: Co-PI, $213,458), Jul 2015 - Jun 2019.
Tobler M, Shaw JH, Kelley J. Physiological Adaptation to Extreme Environments: Genes, Function, and
Evolutionary Patterns. NSF-IOS Integrative Ecological Physiology, total $800,005 (Role: Co-PI, $150,765), Mar 2016 - Mar 2019.
Shaw JH. The role of angiogenic factors in the development of atherosclerosis during Chlamydia pneumoniae infection. Oklahoma Center for Respiratory & Infectious Diseases, NIH-CoBRE, total
$50,000 (Role: PI, $50,000), Jul 2015 – Jun 2016.
Shaw, JH. Advancing the Physiology Curriculum with iWorx animal and human physiology lab teaching
kits. Oklahoma State University Arts & Sciences Student Tech Fee Program (Role: PI, $39,440), 2013 – 2014.
Shaw, JH. Development of a Shared Tissue Culture Facility. Oklahoma State University VP for
Research & Technology Transfer Funds (Role: PI, $59,749), 2012-2013.
Shaw, JH. The Effects of Shear Stress on Endogenous Production of Hydrogen Sulfide by Human
Coronary Artery Smooth Muscle Cells, Oklahoma State University (Role: PI, $6,500), 2013.
Shaw, JH. Effect of hyperlipidemia on signaling pathways mediating exercise-induced vascular
remodeling. Research Advisory Committee (RAC) Award, College of Veterinary Health
Sciences, Oklahoma State University (Role: PI, $9,947), 2010-11.